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INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, 20(22) [10.3390/ijms20225781].";s:4:"data";s:4:"2019";s:2:"id";s:20:"PUBBLICAZIONE_379027";s:6:"handle";s:11:"2108/238769";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore BIO/10";s:9:"metadata4";N;s:9:"metadata5";s:65:"p63 Is a Promising Marker in the Diagnosis of Unusual Skin Cancer";s:9:"metadata6";s:95:"Smirnov, A; Anemona, L; Novelli, F; Piro, MC; Annicchiarico-Petruzzelli, M; Melino, G; Candi, E";s:9:"metadata7";s:20:"10.3390/ijms20225781";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:21;a:14:{s:9:"citazione";s:231:"Smirnov, A., Lena, A.M., Cappello, A., Panatta, E., Anemona, L., Bischetti, S., et al. (2019). ZNF185 is a p63 target gene critical for epidermal differentiation and squamous cell carcinoma development. ONCOGENE, 38(10), 1625-1638.";s:4:"data";s:4:"2019";s:2:"id";s:20:"PUBBLICAZIONE_379026";s:6:"handle";s:11:"2108/238776";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore BIO/10";s:9:"metadata4";N;s:9:"metadata5";s:106:"ZNF185 is a p63 target gene critical for epidermal differentiation and squamous cell carcinoma development";s:9:"metadata6";s:136:"Smirnov, A; Lena, AM; Cappello, A; Panatta, E; Anemona, L; Bischetti, S; Annicchiarico-Petruzzelli, M; Mauriello, A; Melino, G; Candi, E";s:9:"metadata7";s:25:"10.1038/s41388-018-0509-4";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:22;a:14:{s:9:"citazione";s:216:"Frezza, V., Fierro, C., Gatti, E., Peschiaroli, A., Lena, A., Petruzzelli, M., et al. (2018). ΔNp63 promotes IGF1 signalling through IRS1 in squamous cell carcinoma. AGING, 10(12), 4224-4240 [10.18632/aging.101725].";s:4:"data";s:7:"2018-12";s:2:"id";s:20:"PUBBLICAZIONE_377126";s:6:"handle";s:11:"2108/236392";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore BIO/11";s:9:"metadata4";N;s:9:"metadata5";s:71:"ΔNp63 promotes IGF1 signalling through IRS1 in squamous cell carcinoma";s:9:"metadata6";s:145:"Frezza, V; Fierro, C; Gatti, E; Peschiaroli, A; Lena, A; Petruzzelli, M; Candi, E; Anemona, L; Mauriello, A; Pelicci, P; Melino, G; Bernassola, F";s:9:"metadata7";s:21:"10.18632/aging.101725";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:23;a:14:{s:9:"citazione";s:223:"Gurnari, C., Franceschini, L., Anemona, L., Passarelli, F., Vaccarini, S., Pupo, L., et al. (2018). Recurrent Sweet's syndrome in a patient with multiple myeloma. CLINICAL CASE REPORTS, 6(10), 1958-1960 [10.1002/ccr3.1764].";s:4:"data";s:4:"2018";s:2:"id";s:20:"PUBBLICAZIONE_384435";s:6:"handle";s:11:"2108/242768";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore MED/15";s:9:"metadata4";N;s:9:"metadata5";s:61:"Recurrent Sweet's syndrome in a patient with multiple myeloma";s:9:"metadata6";s:127:"Gurnari, C; Franceschini, L; Anemona, L; Passarelli, F; Vaccarini, S; Pupo, L; Provenzano, I; Nasso, D; Rizzo, M; Cantonetti, M";s:9:"metadata7";s:17:"10.1002/ccr3.1764";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:24;a:14:{s:9:"citazione";s:252:"Rovella, V., Anemona, L., Cardellini, M., Scimeca, M., Saggini, A., Santeusanio, G., et al. (2018). The role of obesity in carotid plaque instability: Interaction with age, gender, and cardiovascular risk factors. CARDIOVASCULAR DIABETOLOGY, 17(1), 46.";s:4:"data";s:4:"2018";s:2:"id";s:20:"PUBBLICAZIONE_346187";s:6:"handle";s:11:"2108/210941";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:36:"Settore MED/08 - Anatomia Patologica";s:9:"metadata4";s:2029:"Background: In the last decade, several studies have reported an unexpected and seemingly paradoxical inverse correlation between BMI and incidence of cardiovascular diseases. This so called "obesity paradox effect" has been mainly investigated through imaging methods instead of histologic evaluation, which is still the best method to study the instability of carotid plaque. Therefore, the purpose of our study was to evaluate by histology the role of obesity in destabilization of carotid plaques and the interaction with age, gender and other major cerebrovascular risk factors. Methods: A total of 390 carotid plaques from symptomatic and asymptomatic patients submitted to endarterectomy, for whom complete clinical and laboratory assessment of major cardiovascular risk factors was available, were studied by histology. Patients with a BMI ≥ 30.0 kg/m2 were considered as obese. Data were analyzed by multivariate logistic regression and for each variable in the equation the estimated odds ratio (OR) was calculated. Results: Unstable carotid plaque OR for obese patients with age < 70 years was 5.91 (95% CI 1.17-29.80), thus being the highest OR compared to that of other risk factors. Unstable carotid plaque OR decreased to 4.61 (95% CI 0.54-39.19) in males ≥ 70 years, being only 0.93 (95% CI 0.25-3.52) among women. When obesity featured among metabolic syndrome risk factors, the OR for plaque destabilization was 3.97 (95% CI 1.81-6.22), a significantly higher value compared to OR in non-obese individuals with metabolic syndrome (OR = 1.48; 95% CI 0.86-2.31). Similar results were obtained when assessing the occurrence of acute cerebrovascular symptoms. Conclusions: Results from our study appear to do not confirm any paradoxical effect of obesity on the carotid artery district. Conversely, obesity is confirmed to be an independent risk factor for carotid plaque destabilization, particularly in males aged < 70 years, significantly increasing such risk among patients with metabolic syndrome.";s:9:"metadata5";s:112:"The role of obesity in carotid plaque instability: Interaction with age, gender, and cardiovascular risk factors";s:9:"metadata6";s:174:"Rovella, V; Anemona, L; Cardellini, M; Scimeca, M; Saggini, A; Santeusanio, G; Bonanno, E; Montanaro, M; Legramante, IM; Ippoliti, A; Di Daniele, N; Federici, M; Mauriello, A";s:9:"metadata7";s:25:"10.1186/s12933-018-0685-0";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:25;a:14:{s:9:"citazione";s:197:"Smirnov, A., Cappello, A., Lena, A.M., Anemona, L., Mauriello, A., Di Daniele, N., et al. (2018). ZNF185 is a p53 target gene following DNA damage. AGING, 10(11), 3308-3326 [10.18632/aging.101639].";s:4:"data";s:4:"2018";s:2:"id";s:20:"PUBBLICAZIONE_379060";s:6:"handle";s:11:"2108/238061";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore BIO/11";s:9:"metadata4";N;s:9:"metadata5";s:48:"ZNF185 is a p53 target gene following DNA damage";s:9:"metadata6";s:125:"Smirnov, A; Cappello, A; Lena, AM; Anemona, L; Mauriello, A; Di Daniele, N; Annicchiarico-Petruzzelli, M; Melino, G; Candi, E";s:9:"metadata7";s:21:"10.18632/aging.101639";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:26;a:14:{s:9:"citazione";s:289:"Moscato, G., Giacobbi, E., Anemona, L., Di Cesare, S., Di Matteo, G., Andreoni, M., et al. (2017). Dysplasia of Granulocytes in a Patient with HPV Disease, Recurrent Infections, and B Lymphopenia: A Novel Variant of WHIM Syndrome?. FRONTIERS IN PEDIATRICS, 5, 95 [10.3389/fped.2017.00095].";s:4:"data";s:10:"2017-05-02";s:2:"id";s:20:"PUBBLICAZIONE_313356";s:6:"handle";s:11:"2108/182455";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:35:"Settore MED/17 - Malattie Infettive";s:9:"metadata4";N;s:9:"metadata5";s:131:"Dysplasia of Granulocytes in a Patient with HPV Disease, Recurrent Infections, and B Lymphopenia: A Novel Variant of WHIM Syndrome?";s:9:"metadata6";s:113:"Moscato, G; Giacobbi, E; Anemona, L; Di Cesare, S; Di Matteo, G; Andreoni, M; Mauriello, A; Moschese, V; Chini, L";s:9:"metadata7";s:23:"10.3389/fped.2017.00095";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:27;a:14:{s:9:"citazione";s:253:"Bischetti, S., Scimeca, M., Bonanno, E., Federici, M., Anemona, L., Menghini, R., et al. (2017). Carotid plaque unstability is not related to quantity but to elemental composition of calcification. NMCD. NUTRITION METABOLISM AND CARDIOVASCULAR DISEASES.";s:4:"data";s:4:"2017";s:2:"id";s:20:"PUBBLICAZIONE_316418";s:6:"handle";s:11:"2108/185315";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:36:"Settore MED/22 - Chirurgia Vascolare";s:9:"metadata4";N;s:9:"metadata5";s:99:"Carotid plaque unstability is not related to quantity but to elemental composition of calcification";s:9:"metadata6";s:128:"Bischetti, S; Scimeca, M; Bonanno, E; Federici, M; Anemona, L; Menghini, R; Casella, S; Cardellini, M; Ippoliti, A; Mauriello, A";s:9:"metadata7";s:28:"10.1016/j.numecd.2017.05.006";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:28;a:14:{s:9:"citazione";s:299:"Compagnone, M., Gatti, V., Presutti, D., Ruberti, G., Fierro, C., Markert, E.K., et al. (2017). ΔNp63-mediated regulation of hyaluronic acid metabolism and signaling supports HNSCC tumorigenesis. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 114(50), 13254-13259.";s:4:"data";s:4:"2017";s:2:"id";s:20:"PUBBLICAZIONE_337827";s:6:"handle";s:11:"2108/204065";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore BIO/11";s:9:"metadata4";N;s:9:"metadata5";s:99:"ΔNp63-mediated regulation of hyaluronic acid metabolism and signaling supports HNSCC tumorigenesis";s:9:"metadata6";s:169:"Compagnone, M; Gatti, V; Presutti, D; Ruberti, G; Fierro, C; Markert, EK; Vousden, KH; Zhou, H; Mauriello, A; Anemona, L; Bongiorno-Borbone, L; Melino, G; Peschiaroli, A";s:9:"metadata7";s:23:"10.1073/pnas.1711777114";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:29;a:14:{s:9:"citazione";s:256:"Mauriello, A., Giacobbi, E., Saggini, A., Isgro, A., Facchetti, S., & Anemona, L. (2017). Histological features of bone marrow in paediatric patients during the asymptomatic phase of early-stage Black African sickle cell anaemia. PATHOLOGY, 49(3), 297-303.";s:4:"data";s:4:"2017";s:2:"id";s:20:"PUBBLICAZIONE_382884";s:6:"handle";s:11:"2108/241571";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore MED/08";s:9:"metadata4";N;s:9:"metadata5";s:138:"Histological features of bone marrow in paediatric patients during the asymptomatic phase of early-stage Black African sickle cell anaemia";s:9:"metadata6";s:73:"Mauriello, A; Giacobbi, E; Saggini, A; Isgro, A; Facchetti, S; Anemona, L";s:9:"metadata7";s:28:"10.1016/j.pathol.2016.11.010";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:30;a:14:{s:9:"citazione";s:239:"Mauriello, A., Rovella, V., Borri, F., Anemona, L., Giannini, E., Giacobbi, E., et al. (2017). Hypertension in kidney transplantation is associated with an early renal nerve sprouting. NEPHROLOGY DIALYSIS TRANSPLANTATION, 32(6), 1053-1060.";s:4:"data";s:4:"2017";s:2:"id";s:20:"PUBBLICAZIONE_377490";s:6:"handle";s:11:"2108/236699";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore MED/24";s:9:"metadata4";N;s:9:"metadata5";s:88:"Hypertension in kidney transplantation is associated with an early renal nerve sprouting";s:9:"metadata6";s:183:"Mauriello, A; Rovella, V; Borri, F; Anemona, L; Giannini, E; Giacobbi, E; Saggini, A; Palmieri, G; Anselmo, A; Bove, P; Melino, G; Valentina, G; Tesauro, M; Gabriele, D; Di Daniele, N";s:9:"metadata7";s:18:"10.1093/ndt/gfx069";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:31;a:14:{s:9:"citazione";s:239:"Mauriello, A., Rovella, V., Borri, F., Anemona, L., Giannini, E., Giacobbi, E., et al. (2017). Hypertension in kidney transplantation is associated with an early renal nerve sprouting. NEPHROLOGY DIALYSIS TRANSPLANTATION, 32(6), 1053-1060.";s:4:"data";s:4:"2017";s:2:"id";s:20:"PUBBLICAZIONE_388356";s:6:"handle";s:11:"2108/246255";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:14:"Settore MED/09";s:9:"metadata4";N;s:9:"metadata5";s:88:"Hypertension in kidney transplantation is associated with an early renal nerve sprouting";s:9:"metadata6";s:183:"Mauriello, A; Rovella, V; Borri, F; Anemona, L; Giannini, E; Giacobbi, E; Saggini, A; Palmieri, G; Anselmo, A; Bove, P; Melino, G; Valentina, G; Tesauro, M; Gabriele, D; Di Daniele, N";s:9:"metadata7";s:18:"10.1093/ndt/gfx069";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:32;a:14:{s:9:"citazione";s:276:"Postorino, M., Pupo, L., Provenzano, I., Del Principe, M.i., Buccisano, F., Franceschini, L., et al. (2016). A case of oral mycosis fungoides successfully treated by combination of alemtuzumab and chemotherapy. ANNALS OF HEMATOLOGY, 95(1), 153-154 [10.1007/s00277-015-2503-z].";s:4:"data";s:7:"2016-01";s:2:"id";s:20:"PUBBLICAZIONE_258459";s:6:"handle";s:11:"2108/137388";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:36:"Settore MED/15 - Malattie del Sangue";s:9:"metadata4";N;s:9:"metadata5";s:100:"A case of oral mycosis fungoides successfully treated by combination of alemtuzumab and chemotherapy";s:9:"metadata6";s:215:"Postorino, M; Pupo, L; Provenzano, I; Del Principe, Mi; Buccisano, F; Franceschini, L; Rizzo, M; Nasso, D; Meconi, F; Onnis, I; Vaccarini, S; Zizzari, A; Anemona, L; Mauramati, S; Bruno, E; Cantonetti, M; Amadori, S";s:9:"metadata7";s:25:"10.1007/s00277-015-2503-z";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:33;a:14:{s:9:"citazione";s:346:"Mauriello, A., Rovella, V., Anemona, L., Servadei, F., Giannini, E., Bove, P., et al. (2015). Increased Sympathetic Renal Innervation in Hemodialysis Patients Is the Anatomical Substrate of Sympathetic Hyperactivity in End-Stage Renal Disease. JOURNAL OF THE AMERICAN HEART ASSOCIATION. CARDIOVASCULAR AND CEREBROVASCULAR DISEASE, 4(12), e002426.";s:4:"data";s:7:"2015-12";s:2:"id";s:20:"PUBBLICAZIONE_264161";s:6:"handle";s:11:"2108/143607";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:25:"Settore MED/24 - Urologia";s:9:"metadata4";s:1663:"Background: Renal denervation represents an emerging treatment for resistant hypertension in patients with end-stage renal disease, but data about the anatomic substrate of this treatment are lacking. Therefore, the aim of this study was to investigate the morphological basis of sympathetic hyperactivity in the setting of hemodialysis patients to identify an anatomical substrate that could warrant the use of this new therapeutic approach. Methods and results: The distribution of sympathetic nerves was evaluated in the adventitia of 38 renal arteries that were collected at autopsy or during surgery from 25 patients: 9 with end-stage renal disease on dialysis (DIAL group) and 16 age-matched control nondialysis patients (CTRL group). Patients in the DIAL group showed a significant increase in nerve density in the internal area of the peri-adventitial tissue (within the first 0.5 mm of the beginning of the adventitia) compared with the CTRL group (4.01±0.30 versus 2.87±0.28×mm(2), P=0.01). Regardless of dialysis, hypertensive patients with signs of severe arteriolar damage had a greater number of nerve endings in the most internal adventitia, and this number was significantly higher than in patients without hypertensive arteriolar damage (3.90±0.36 versus 2.87±0.41×mm(2), P=0.04), showing a correlation with hypertensive arteriolar damage rather than with hypertensive clinical history. Conclusions: The findings from this study provide a morphological basis underlying sympathetic hyperactivity in patients with end-stage renal disease and might offer useful information to improve the use of renal denervation in this group of patients.";s:9:"metadata5";s:148:"Increased Sympathetic Renal Innervation in Hemodialysis Patients Is the Anatomical Substrate of Sympathetic Hyperactivity in End-Stage Renal Disease";s:9:"metadata6";s:109:"Mauriello, A; Rovella, V; Anemona, L; Servadei, F; Giannini, E; Bove, P; Anselmo, A; Melino, G; Di Daniele, N";s:9:"metadata7";s:23:"10.1161/JAHA.115.002426";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:34;a:14:{s:9:"citazione";s:328:"Menghini, R., Casagrande, V., Cardellini, M., Ballanti, M., Davato, F., Cardolini, I., et al. (2015). FoxO1 regulates asymmetric dimethylarginine via downregulation of dimethylaminohydrolase 1 in human endothelial cells and subjects with atherosclerosis. ATHEROSCLEROSIS, 242(1), 230-235 [10.1016/j.atherosclerosis.2015.07.026].";s:4:"data";s:4:"2015";s:2:"id";s:20:"PUBBLICAZIONE_235755";s:6:"handle";s:11:"2108/130009";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:54:"Settore MED/49 - Scienze Tecniche Dietetiche Applicate";s:9:"metadata4";N;s:9:"metadata5";s:151:"FoxO1 regulates asymmetric dimethylarginine via downregulation of dimethylaminohydrolase 1 in human endothelial cells and subjects with atherosclerosis";s:9:"metadata6";s:203:"Menghini, R; Casagrande, V; Cardellini, M; Ballanti, M; Davato, F; Cardolini, I; Stoehr, R; Fabrizi, M; Morelli, M; Anemona, L; Bernges, I; Schwedhelm, E; Ippoliti, A; Mauriello, A; Boger, R; Federici, M";s:9:"metadata7";s:37:"10.1016/j.atherosclerosis.2015.07.026";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:35;a:14:{s:9:"citazione";s:234:"Sancesario, G.M., Schirinzi, T., Anemona, L., Pisani, A., Porzio, O., Sancesario, G., et al. (2015). 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