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JOURNAL OF CLINICAL DENSITOMETRY, 12(1), 112.";s:4:"data";s:4:"2009";s:2:"id";s:19:"PUBBLICAZIONE_94420";s:6:"handle";s:10:"2108/41931";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:45:"Settore MED/33 - Malattie Apparato Locomotore";s:9:"metadata4";N;s:9:"metadata5";s:69:"Prevalence of Osteopenia and Osteoporosis in Italian Women Population";s:9:"metadata6";s:54:"Andreoli, A; Lauro, D; Celi, M; Sorge, R; Tarantino, U";s:9:"metadata7";s:26:"10.1016/j.jocd.2008.07.043";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:134;a:14:{s:9:"citazione";s:279:"Bellia, A., Giardina, E., Lauro, D., Tesauro, M., Di Fede, G., Cusumano, G., et al. (2009). "The Linosa Study": Epidemiological and heritability data of the metabolic syndrome in a Caucasian genetic isolate. NMCD. NUTRITION METABOLISM AND CARDIOVASCULAR DISEASES, 19(7), 455-461.";s:4:"data";s:4:"2009";s:2:"id";s:20:"PUBBLICAZIONE_240055";s:6:"handle";s:11:"2108/121189";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:264:"Adolescent; Adult; Age Factors; Aged; Blood Glucose; Cholesterol, HDL; European Continental Ancestry Group; Female; Genetic Linkage; Humans; Hypertriglyceridemia; Insulin Resistance; Italy; Male; Metabolic Syndrome X; Middle Aged; Sex Factors; Smoking; Young Adult";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:377:"Growing evidence suggests that the metabolic syndrome (MetS) has both a genetic and environmental basis. To evaluate the possibility of a further genetic analysis, we estimated prevalence rates and heritabilities for the MetS and its individual traits in the adult population of Linosa, a small and isolated Italian Island in the southern-central part of the Mediterranean Sea.";s:9:"metadata5";s:114:""The Linosa Study": Epidemiological and heritability data of the metabolic syndrome in a Caucasian genetic isolate";s:9:"metadata6";s:126:"Bellia, A; Giardina, E; Lauro, D; Tesauro, M; Di Fede, G; Cusumano, G; Federici, M; Rini, G; Novelli, G; Lauro, R; Sbraccia, P";s:9:"metadata7";s:28:"10.1016/j.numecd.2008.11.002";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:135;a:14:{s:9:"citazione";s:238:"Rizza, S., Tesauro, M., Cardillo, C., Galli, A., Iantorno, M., Gigli, F., et al. (2009). Fish oil supplementation improves endothelial function in normoglycemic offspring of patients with type 2 diabetes. ATHEROSCLEROSIS, 206(2), 569-574.";s:4:"data";s:4:"2009";s:2:"id";s:20:"PUBBLICAZIONE_240059";s:6:"handle";s:11:"2108/121191";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:200:"Adult; Blood Glucose; Diabetes Mellitus, Type 2; Double-Blind Method; Endothelium; Fatty Acids, Omega-3; Female; Fish Oils; Genetic Predisposition to Disease; Humans; Male; Tumor Necrosis Factor-alpha";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:451:"Offspring of patients with type 2 diabetes (OPDs) exhibits endothelial dysfunction (ED) associated with a chronic inflammatory state. N-3 polyunsaturated fatty acids (n-3 PUFA) may have antioxidant and anti-inflammatory properties that are beneficial for cardiovascular and metabolic health. Therefore, in the present study, we tested the hypothesis that dietary supplementation with fish oil rich in n-3 PUFA may improve ED in otherwise healthy OPDs.";s:9:"metadata5";s:114:"Fish oil supplementation improves endothelial function in normoglycemic offspring of patients with type 2 diabetes";s:9:"metadata6";s:112:"Rizza, S; Tesauro, M; Cardillo, C; Galli, A; Iantorno, M; Gigli, F; Sbraccia, P; Federici, M; Quon, MJ; Lauro, D";s:9:"metadata7";s:37:"10.1016/j.atherosclerosis.2009.03.006";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:136;a:14:{s:9:"citazione";s:232:"Tesauro, M., Schinzari, F., Rovella, V., Di Daniele, N., Lauro, D., Mores, N., et al. (2009). Ghrelin restores the endothelin 1/nitric oxide balance in patients with obesity-related metabolic syndrome. HYPERTENSION, 54(5), 995-1000.";s:4:"data";s:4:"2009";s:2:"id";s:20:"PUBBLICAZIONE_240078";s:6:"handle";s:11:"2108/121210";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:352:"Adult; Analysis of Variance; Blood Flow Velocity; Case-Control Studies; Endothelium, Vascular; Female; Forearm; Ghrelin; Homeostasis; Humans; Male; Metabolic Syndrome X; Middle Aged; Nitric Oxide Synthase; Obesity; Plethysmography; Probability; Receptor, Endothelin A; Reference Values; Vasoconstriction; Vasodilation; Endothelin A Receptor Antagonists";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:1839:"Obesity is associated with endothelial dysfunction related to decreased NO bioavailability, increased endothelin 1 vasoconstrictor activity, and decreased circulating ghrelin. Therefore, we tested whether exogenous ghrelin may have benefits to improve the balance between endothelin 1 and NO in patients with obesity-related metabolic syndrome. Vasoactive actions of endothelin 1 and NO were assessed in 8 patients with metabolic syndrome and 8 matched controls by evaluating forearm blood flow responses (strain-gauge plethysmography) to intra-arterial infusion of BQ-123 (endothelin A receptor antagonist; 10 nmol/min), followed by NG-monomethyl-L-arginine (NO synthase inhibitor; 4 micromol/min), before and after infusion of ghrelin (200 ng/min). In the absence of ghrelin, the vasodilator response to BQ-123 was greater in patients than in controls (P<0.001), whereas infusion of NG-monomethyl-L-arginine induced smaller vasoconstriction in patients than in controls (P=0.006). Importantly, exogenous ghrelin decreased the vasodilator response to BQ-123 (P=0.007 versus saline) and enhanced the magnitude of changes in forearm blood flow induced by NG-monomethyl-L-arginine (P=0.003) in patients but not in controls (both P>0.05). The favorable effect of ghrelin on endothelin A-dependent vasoconstriction was likely related to the stimulation of NO production, because no change in the vascular effect of BQ-123 was observed after ghrelin (P=0.44) in 5 patients with metabolic syndrome during continuous infusion of the NO donor sodium nitroprusside (0.2 microg/min). In patients with metabolic syndrome, ghrelin has benefits to normalize the balance between vasoconstrictor (endothelin 1) and vasodilating (NO) mediators, thus suggesting that this peptide has important peripheral actions to preserve vascular homeostasis in humans.";s:9:"metadata5";s:106:"Ghrelin restores the endothelin 1/nitric oxide balance in patients with obesity-related metabolic syndrome";s:9:"metadata6";s:98:"Tesauro, M; Schinzari, F; Rovella, V; Di Daniele, N; Lauro, D; Mores, N; Veneziani, A; Cardillo, C";s:9:"metadata7";s:34:"10.1161/HYPERTENSIONAHA.109.137729";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:137;a:14:{s:9:"citazione";s:253:"Hribal, M., Tornei, F., Pujol, A., Menghini, R., Barcaroli, D., Lauro, D., et al. (2008). Transgenic mice overexpressing human G972R IRS-1 show impaired insulin action and insulin secretion. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE, 12(5B), 2096-2106.";s:4:"data";s:7:"2008-10";s:2:"id";s:19:"PUBBLICAZIONE_88816";s:6:"handle";s:10:"2108/41889";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:62:"Settore M-EDF/01 - Metodi e Didattiche delle Attivita' Motorie";s:9:"metadata4";N;s:9:"metadata5";s:99:"Transgenic mice overexpressing human G972R IRS-1 show impaired insulin action and insulin secretion";s:9:"metadata6";s:122:"Hribal, M; Tornei, F; Pujol, A; Menghini, R; Barcaroli, D; Lauro, D; Amoruso, R; Lauro, R; Bosch, F; Sesti, G; Federici, M";s:9:"metadata7";s:32:"10.1111/j.1582-4934.2008.00246.x";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:138;a:14:{s:9:"citazione";s:231:"Tesauro, M., Schinzari, F., Rovella, V., Melina, D., Mores, N., Barini, A., et al. (2008). Tumor necrosis factor-alpha antagonism improves vasodilation during hyperinsulinemia in metabolic syndrome. DIABETES CARE, 31(7), 1439-1441.";s:4:"data";s:7:"2008-07";s:2:"id";s:19:"PUBBLICAZIONE_90176";s:6:"handle";s:10:"2108/41892";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:31:"Settore MED/13 - Endocrinologia";s:9:"metadata4";N;s:9:"metadata5";s:106:"Tumor necrosis factor-alpha antagonism improves vasodilation during hyperinsulinemia in metabolic syndrome";s:9:"metadata6";s:127:"Tesauro, M; Schinzari, F; Rovella, V; Melina, D; Mores, N; Barini, A; Mettimano, M; Lauro, D; Iantorno, M; Quon, M; Cardillo, C";s:9:"metadata7";s:17:"10.2337/dc08-0219";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:139;a:14:{s:9:"citazione";s:323:"Scuteri, A., Tesauro, M., Rizza, S., Iantorno, M., Federici, M., Lauro, D., et al. (2008). Endothelial function and arterial stiffness in normotensive normoglycemic first-degree relatives of diabetic patients are independent of the metabolic syndrome. NMCD. NUTRITION METABOLISM AND CARDIOVASCULAR DISEASES, 18(5), 349-356.";s:4:"data";s:7:"2008-06";s:2:"id";s:19:"PUBBLICAZIONE_90216";s:6:"handle";s:10:"2108/41904";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:159:"Endothelial function and arterial stiffness in normotensive normoglycemic first-degree relatives of diabetic patients are independent of the metabolic syndrome";s:9:"metadata6";s:130:"Scuteri, A; Tesauro, M; Rizza, S; Iantorno, M; Federici, M; Lauro, D; Campia, U; Turriziani, M; Fusco, A; Cocciolillo, G; Lauro, R";s:9:"metadata7";s:28:"10.1016/j.numecd.2007.03.008";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:140;a:14:{s:9:"citazione";s:194:"Marchetti, P., Dotta, F., Lauro, D., & Purrello, F. (2008). An overview of pancreatic beta-cell defects in human type 2 diabetes: implications for treatment. REGULATORY PEPTIDES, 146(1-3), 4-11.";s:4:"data";s:10:"2008-02-07";s:2:"id";s:19:"PUBBLICAZIONE_89941";s:6:"handle";s:10:"2108/41910";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:31:"Settore MED/13 - Endocrinologia";s:9:"metadata4";N;s:9:"metadata5";s:96:"An overview of pancreatic beta-cell defects in human type 2 diabetes: implications for treatment";s:9:"metadata6";s:45:"Marchetti, P; Dotta, F; Lauro, D; Purrello, F";s:9:"metadata7";s:28:"10.1016/j.regpep.2007.08.017";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:141;a:14:{s:9:"citazione";s:326:"Scuteri, A., Cocciolillo, G., Lauro, R., Tesauro, M., Rizza, S., Iantorno, M., et al. (2008). Endothelial function and arterial stiffness in normotensive normoglycemic first-degree relatives of diabetic patients are independent of the metabolic syndrome. NMCD. NUTRITION METABOLISM AND CARDIOVASCULAR DISEASES, 18(5), 349-356.";s:4:"data";s:4:"2008";s:2:"id";s:20:"PUBBLICAZIONE_240049";s:6:"handle";s:11:"2108/121181";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:159:"Endothelial function and arterial stiffness in normotensive normoglycemic first-degree relatives of diabetic patients are independent of the metabolic syndrome";s:9:"metadata6";s:130:"Scuteri, A; Cocciolillo, G; Lauro, R; Tesauro, M; Rizza, S; Iantorno, M; Federici, M; Lauro, D; Campia, U; Turriziani, M; Fusco, A";s:9:"metadata7";s:28:"10.1016/j.numecd.2007.03.008";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:142;a:14:{s:9:"citazione";s:228:"Tesauro, M., Schinzari, F., Rovella, V., Melina, D., Mores, N., Barini, A., et al. (2008). Tumor necrosis factor-α antagonism improves vasodilation during hyperinsulinemia in metabolic syndrome. DIABETES CARE, 31(7), 1439-1441.";s:4:"data";s:4:"2008";s:2:"id";s:20:"PUBBLICAZIONE_240053";s:6:"handle";s:11:"2108/121185";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:251:"Acetylcholine; Analysis of Variance; Anti-Inflammatory Agents; Antibodies, Monoclonal; Blood Glucose; Cholesterol; Humans; Hyperinsulinism; Metabolic Syndrome X; Nitroprusside; Reference Values; Triglycerides; Tumor Necrosis Factor-alpha; Vasodilation";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:296:"Obesity is associated with chronic inflammation due to overproduction of proinflammatory cytokines, including tumor necrosis factor (TNF)-alpha. We assessed the effects of TNF-alpha neutralization by infliximab on vascular reactivity during hyperinsulinemia in obesity-related metabolic syndrome.";s:9:"metadata5";s:103:"Tumor necrosis factor-α antagonism improves vasodilation during hyperinsulinemia in metabolic syndrome";s:9:"metadata6";s:128:"Tesauro, M; Schinzari, F; Rovella, V; Melina, D; Mores, N; Barini, A; Mettimano, M; Lauro, D; Iantorno, M; Quon, Mj; Cardillo, C";s:9:"metadata7";s:17:"10.2337/dc08-0219";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:143;a:14:{s:9:"citazione";s:260:"Serino, M., Menghini, R., Fiorentino, L., Amoruso, R., Mauriello, A., Lauro, D., et al. (2007). Mice heterozygous for tumor necrosis factor-alpha converting enzyme are protected from obesity-induced insulin resistance and diabetes. DIABETES, 56(10), 2541-2546.";s:4:"data";s:7:"2007-10";s:2:"id";s:19:"PUBBLICAZIONE_91977";s:6:"handle";s:10:"2108/37049";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:134:"Mice heterozygous for tumor necrosis factor-alpha converting enzyme are protected from obesity-induced insulin resistance and diabetes";s:9:"metadata6";s:120:"Serino, M; Menghini, R; Fiorentino, L; Amoruso, R; Mauriello, A; Lauro, D; Sbraccia, P; Hribal, M; Lauro, R; Federici, M";s:9:"metadata7";s:17:"10.2337/db07-0360";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:144;a:14:{s:9:"citazione";s:307:"Iantorno, M., Chen, H., Kim, J., Tesauro, M., Lauro, D., Cardillo, C., et al. (2007). Ghrelin has novel vascular actions that mimic PI 3-kinase-dependent actions of insulin to stimulate production of NO from endothelial cells. AMERICAN JOURNAL OF PHYSIOLOGY: ENDOCRINOLOGY AND METABOLISM, 292(3), E756-E764.";s:4:"data";s:7:"2007-03";s:2:"id";s:19:"PUBBLICAZIONE_95299";s:6:"handle";s:10:"2108/41883";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:139:"Ghrelin has novel vascular actions that mimic PI 3-kinase-dependent actions of insulin to stimulate production of NO from endothelial cells";s:9:"metadata6";s:72:"Iantorno, M; Chen, H; Kim, J; Tesauro, M; Lauro, D; Cardillo, C; Quon, M";s:9:"metadata7";s:26:"10.1152/ajpendo.00570.2006";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:145;a:14:{s:9:"citazione";s:266:"Tesauro, M., Rizza, S., Iantorno, M., Campia, U., Cardillo, C., Lauro, D., et al. (2007). Vascular, metabolic, and inflammatory abnormalities in normoglycemic offspring of patients with type 2 diabetes mellitus. METABOLISM, CLINICAL AND EXPERIMENTAL, 56(3), 413-419.";s:4:"data";s:7:"2007-03";s:2:"id";s:19:"PUBBLICAZIONE_95311";s:6:"handle";s:10:"2108/41868";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:31:"Settore MED/13 - Endocrinologia";s:9:"metadata4";N;s:9:"metadata5";s:120:"Vascular, metabolic, and inflammatory abnormalities in normoglycemic offspring of patients with type 2 diabetes mellitus";s:9:"metadata6";s:170:"Tesauro, M; Rizza, S; Iantorno, M; Campia, U; Cardillo, C; Lauro, D; Leo, R; Turriziani, M; Cocciolillo, G; Fusco, A; Panza, J; Scuteri, A; Federici, M; Lauro, R; Quon, M";s:9:"metadata7";s:29:"10.1016/j.metabol.2006.10.026";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:146;a:14:{s:9:"citazione";s:337:"Cardellini, M., Marini, M., Frontoni, S., Hribal, M., Andreozzi, F., Perticone, F., et al. (2007). Carotid artery intima-media thickness is associated with insulin-mediated glucose disposal in nondiabetic normotensive offspring of type 2 diabetic patients. AMERICAN JOURNAL OF PHYSIOLOGY: ENDOCRINOLOGY AND METABOLISM, 292(1), E347-E352.";s:4:"data";s:7:"2007-01";s:2:"id";s:19:"PUBBLICAZIONE_89944";s:6:"handle";s:10:"2108/41873";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:31:"Settore MED/13 - Endocrinologia";s:9:"metadata4";N;s:9:"metadata5";s:156:"Carotid artery intima-media thickness is associated with insulin-mediated glucose disposal in nondiabetic normotensive offspring of type 2 diabetic patients";s:9:"metadata6";s:109:"Cardellini, M; Marini, M; Frontoni, S; Hribal, M; Andreozzi, F; Perticone, F; Federici, M; Lauro, D; Sesti, G";s:9:"metadata7";s:26:"10.1152/ajpendo.00291.2006";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:147;a:14:{s:9:"citazione";s:264:"Iantorno, M., Chen, H., Kim, J., Tesauro, M., Lauro, D., Cardillo, C., et al. (2007). Ghrelin has novel vascular actions that mimic PI 3-kinase-dependent actions of insulin to stimulate production of NO from endothelial cells. ENDOCRINOLOGY AND METABOLISM, 292(3).";s:4:"data";s:4:"2007";s:2:"id";s:20:"PUBBLICAZIONE_240035";s:6:"handle";s:11:"2108/121166";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:139:"Ghrelin has novel vascular actions that mimic PI 3-kinase-dependent actions of insulin to stimulate production of NO from endothelial cells";s:9:"metadata6";s:73:"Iantorno, M; Chen, H; Kim, J; Tesauro, M; Lauro, D; Cardillo, C; Quon, MJ";s:9:"metadata7";s:26:"10.1152/ajpendo.00570.2006";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:148;a:14:{s:9:"citazione";s:266:"Tesauro, M., Rizza, S., Iantorno, M., Campia, U., Cardillo, C., Lauro, D., et al. (2007). Vascular, metabolic, and inflammatory abnormalities in normoglycemic offspring of patients with type 2 diabetes mellitus. METABOLISM, CLINICAL AND EXPERIMENTAL, 56(3), 413-419.";s:4:"data";s:4:"2007";s:2:"id";s:20:"PUBBLICAZIONE_240039";s:6:"handle";s:11:"2108/121170";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:120:"Vascular, metabolic, and inflammatory abnormalities in normoglycemic offspring of patients with type 2 diabetes mellitus";s:9:"metadata6";s:173:"Tesauro, M; Rizza, S; Iantorno, M; Campia, U; Cardillo, C; Lauro, D; Leo, R; Turriziani, M; Cocciolillo, Gc; Fusco, A; Panza, JA; Scuteri, A; Federici, M; Lauro, R; Quon, MJ";s:9:"metadata7";s:29:"10.1016/j.metabol.2006.10.026";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:149;a:14:{s:9:"citazione";s:239:"Marchetti, V., Menghini, R., Rizza, S., Vivanti, A., Feccia, T., Lauro, D., et al. (2006). Benfotiamine counteracts glucose toxicity effects on endothelial progenitor cell differentiation via Akt/FoxO signaling. DIABETES, 55(8), 2231-2237.";s:4:"data";s:7:"2006-08";s:2:"id";s:19:"PUBBLICAZIONE_89943";s:6:"handle";s:10:"2108/40819";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:119:"Benfotiamine counteracts glucose toxicity effects on endothelial progenitor cell differentiation via Akt/FoxO signaling";s:9:"metadata6";s:104:"Marchetti, V; Menghini, R; Rizza, S; Vivanti, A; Feccia, T; Lauro, D; Fukamizu, A; Lauro, R; Federici, M";s:9:"metadata7";s:17:"10.2337/db06-0369";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:150;a:14:{s:9:"citazione";s:342:"Rizza, S., Tesauro, M., Cardellini, M., Menghini, R., Bellia, A., Marini, M., et al. (2006). Insulin resistance and increased intimal medial thickness in glucose tolerant offspring of type 2 diabetic subjects carrying the D298D genotype of endothelial nitric oxide synthase. ARTERIOSCLEROSIS, THROMBOSIS, AND VASCULAR BIOLOGY, 26(2), 431-432.";s:4:"data";s:7:"2006-02";s:2:"id";s:19:"PUBBLICAZIONE_89412";s:6:"handle";s:10:"2108/40467";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:180:"Insulin resistance and increased intimal medial thickness in glucose tolerant offspring of type 2 diabetic subjects carrying the D298D genotype of endothelial nitric oxide synthase";s:9:"metadata6";s:126:"Rizza, S; Tesauro, M; Cardellini, M; Menghini, R; Bellia, A; Marini, M; Lauro, D; Sbraccia, P; Sesti, G; Lauro, R; Federici, M";s:9:"metadata7";s:34:"10.1161/01.ATV.0000196552.84475.fc";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:151;a:14:{s:9:"citazione";s:344:"Rizza, S., Tesauro, M., Cardellini, M., Menghini, R., Bellia, A., Marini, M.A., et al. (2006). Insulin resistance and increased intimal medial thickness in glucose tolerant offspring of type 2 diabetic subjects carrying the D298D genotype of endothelial nitric oxide synthase. ARTERIOSCLEROSIS, THROMBOSIS, AND VASCULAR BIOLOGY, 26(2), 431-432.";s:4:"data";s:7:"2006-02";s:2:"id";s:19:"PUBBLICAZIONE_94481";s:6:"handle";s:10:"2108/41005";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:180:"Insulin resistance and increased intimal medial thickness in glucose tolerant offspring of type 2 diabetic subjects carrying the D298D genotype of endothelial nitric oxide synthase";s:9:"metadata6";s:127:"Rizza, S; Tesauro, M; Cardellini, M; Menghini, R; Bellia, A; Marini, MA; Lauro, D; Sbraccia, P; Sesti, G; Lauro, R; Federici, M";s:9:"metadata7";s:34:"10.1161/01.ATV.0000196552.84475.fc";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:152;a:14:{s:9:"citazione";s:232:"Federici, M., Menghini, R., Perego, L., Martelli, E., lauro, D., Porzio, O., et al. (2006). Reduced TIMP3 levels in atherosclerotic plaques from patients affected by altered glucose tolerance. DIABETES, 55(Supplement: 1), A156-A156.";s:4:"data";s:4:"2006";s:2:"id";s:20:"PUBBLICAZIONE_141224";s:6:"handle";s:10:"2108/68713";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:36:"Settore MED/08 - Anatomia Patologica";s:9:"metadata4";N;s:9:"metadata5";s:99:"Reduced TIMP3 levels in atherosclerotic plaques from patients affected by altered glucose tolerance";s:9:"metadata6";s:118:"Federici, M; Menghini, R; Perego, L; Martelli, E; lauro, D; Porzio, O; Mauriello, A; Pontiroli, AE; Lauro, R; Folli, F";s:9:"metadata7";N;s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:153;a:14:{s:9:"citazione";s:249:"Iellamo, F., Tesauro, M., Rizza, S., Aquilani, S., Cardillo, C., Iantorno, M., et al. (2006). Concomitant impairment in endothelial function and neural cardiovascular regulation in offspring of type 2 diabetic subjects. HYPERTENSION, 48(3), 418-423.";s:4:"data";s:4:"2006";s:2:"id";s:20:"PUBBLICAZIONE_240031";s:6:"handle";s:11:"2108/121162";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:225:"Adult; Autonomic Nervous System; Baroreflex; Brachial Artery; Cardiovascular System; Case-Control Studies; Diabetes Mellitus, Type 2; Endothelium, Vascular; Female; Humans; Male; Regional Blood Flow; Vagus Nerve; Vasodilation";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:1721:"Endothelial function is impaired in first-degree relatives (FDRs) of patients with type 2 diabetes. Many states characterized by endothelial dysfunction are associated with increased cardiovascular sympathetic outflow. In this study, we investigated endothelial and autonomic nervous system (ANS) functioning in FDRs and tested the hypothesis that in basal condition, impaired endothelial function is associated with impaired cardiovascular ANS regulation. Flow-mediated endothelium-dependent and -independent vasodilation of the brachial artery was measured with high-resolution ultrasound in 27 otherwise healthy FDRs (14 men and 13 women; mean age 32 years) with normal oral glucose tolerance and in 15 age- and gender-matched control subjects. Cardiovascular ANS regulation was investigated by means of spectral analysis of heart rate and systolic blood pressure (SBP) variability. Baroreflex sensitivity was assessed by the spontaneous baroreflex sequences technique. Flow-mediated endothelium-dependent vasodilation was 9.4+/-1.0% in FDRs and 17.0+/-2.3% in control subjects (P=0.001). Low-frequency oscillations in SBP variability were 8.6+/-2.8 and 2.8+/-0.6 mm Hg in FDRs and controls, respectively (P=0.04). Baroreflex sensitivity was significantly less in FDRs than controls (22.8+/-2.7 versus 37.0+/-5.8, respectively; P=0.01). Change in vessel diameter was inversely correlated with the low-frequency component of SBP variability (r=-0.40; P=0.014). In healthy FDRs of diabetic patients there is a concomitant, possibly related, impairment in endothelial and ANS functioning, which manifests, indirectly, with increase in vascular sympathetic outflow and a depressed baroreflex, vagal, control of heart rate.";s:9:"metadata5";s:124:"Concomitant impairment in endothelial function and neural cardiovascular regulation in offspring of type 2 diabetic subjects";s:9:"metadata6";s:96:"Iellamo, F; Tesauro, M; Rizza, S; Aquilani, S; Cardillo, C; Iantorno, M; Turriziani, M; Lauro, R";s:9:"metadata7";s:34:"10.1161/01.HYP.0000234648.62994.ab";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:154;a:14:{s:9:"citazione";s:190:"Leo, R., Di Lorenzo, G., Tesauro, M., Cola, C., Fortuna, E., Zanasi, M., et al. (2006). Decreased plasma adiponectin concentration in major depression. NEUROSCIENCE LETTERS, 407(3), 211-213.";s:4:"data";s:4:"2006";s:2:"id";s:20:"PUBBLICAZIONE_240033";s:6:"handle";s:11:"2108/121164";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:95:"Adiponectin; Adult; Depressive Disorder, Major; Female; Humans; Male; Severity of Illness Index";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:832:"Adiponectin is the most abundant adipose-derived plasma protein. Recently adiponectin levels have been linked to most variables of metabolic syndrome and conventional risk factors for cardiovascular disease. However, its relation with major depression is yet unclear. We evaluated plasma adiponectin levels in 32 first-episode drug-naïve major depression (DSM-IV-TR) patients without conventional risk factors for cardiovascular disease and 32 matched healthy subjects. Major depression patients displayed lower adiponectin plasma levels compared to controls (P<0.01). Adiponectin significantly correlated with depression severity, as assessed by HAM-D (rho=0.83, P<0.001). This study shows decreased plasma adiponectin concentrations in major depression patients and relates adiponectinemia reduction to major depression severity.";s:9:"metadata5";s:62:"Decreased plasma adiponectin concentration in major depression";s:9:"metadata6";s:111:"Leo, R; Di Lorenzo, G; Tesauro, M; Cola, C; Fortuna, E; Zanasi, M; Troisi, A; Siracusano, A; Lauro, R; Romeo, F";s:9:"metadata7";s:28:"10.1016/j.neulet.2006.08.043";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:155;a:14:{s:9:"citazione";s:344:"Rizza, S., Tesauro, M., Cardellini, M., Menghini, R., Bellia, A., Marini, M.A., et al. (2006). Insulin resistance and increased intimal medial thickness in glucose tolerant offspring of type 2 diabetic subjects carrying the D298D genotype of endothelial nitric oxide synthase. ARTERIOSCLEROSIS, THROMBOSIS, AND VASCULAR BIOLOGY, 26(2), 431-432.";s:4:"data";s:4:"2006";s:2:"id";s:20:"PUBBLICAZIONE_239704";s:6:"handle";s:11:"2108/120837";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:180:"Insulin resistance and increased intimal medial thickness in glucose tolerant offspring of type 2 diabetic subjects carrying the D298D genotype of endothelial nitric oxide synthase";s:9:"metadata6";s:127:"Rizza, S; Tesauro, M; Cardellini, M; Menghini, R; Bellia, A; Marini, MA; Lauro, D; Sbraccia, P; Sesti, G; Lauro, R; Federici, M";s:9:"metadata7";s:34:"10.1161/01.ATV.0000196552.84475.fc";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:156;a:14:{s:9:"citazione";s:304:"Menghini, R., Marchetti, V., Cardellini, M., Hribal, M., Mauriello, A., Lauro, D., et al. (2005). Phosphorylation of GATA2 by Akt increases adipose tissue differentiation and reduces adipose tissue-related inflammation: a novel pathway linking obesity to atherosclerosis. CIRCULATION, 111(15), 1946-1953.";s:4:"data";s:10:"2005-04-19";s:2:"id";s:19:"PUBBLICAZIONE_93007";s:6:"handle";s:10:"2108/36965";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:36:"Settore MED/08 - Anatomia Patologica";s:9:"metadata4";N;s:9:"metadata5";s:172:"Phosphorylation of GATA2 by Akt increases adipose tissue differentiation and reduces adipose tissue-related inflammation: a novel pathway linking obesity to atherosclerosis";s:9:"metadata6";s:111:"Menghini, R; Marchetti, V; Cardellini, M; Hribal, M; Mauriello, A; Lauro, D; Sbraccia, P; Lauro, R; Federici, M";s:9:"metadata7";s:34:"10.1161/01.CIR.0000161814.02942.B2";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:157;a:14:{s:9:"citazione";s:280:"Federici, M., Hribal, M., Menghini, R., Kanno, H., Marchetti, V., Porzio, O., et al. (2005). Timp3 deficiency in insulin receptor-haploinsufficient mice promotes diabetes and vascular inflammation via increased TNF-alpha. THE JOURNAL OF CLINICAL INVESTIGATION, 115(12), 3494-3505.";s:4:"data";s:4:"2005";s:2:"id";s:18:"PUBBLICAZIONE_8759";s:6:"handle";s:10:"2108/40280";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:127:"Timp3 deficiency in insulin receptor-haploinsufficient mice promotes diabetes and vascular inflammation via increased TNF-alpha";s:9:"metadata6";s:225:"Federici, M; Hribal, M.L; Menghini, R; Kanno, H; Marchetti, V; Porzio, O; Sunnarborg, S.W; Rizza, S; Serino, M; Cunsolo, V; Lauro, D; Mauriello, A; Smookler, D.S; Sbraccia, P; Sesti, G; Lee, D.C; Khokha, R; Accili, D Lauro, R";s:9:"metadata7";s:16:"10.1172/JCI26052";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:158;a:14:{s:9:"citazione";s:198:"Tesauro, M., Schinzari, F., Iantorno, M., Rizza, S., Melina, D., Lauro, D., et al. (2005). Ghrelin improves endothelial function in patients with metabolic syndrome. CIRCULATION, 112(19), 2986-2992.";s:4:"data";s:4:"2005";s:2:"id";s:19:"PUBBLICAZIONE_15217";s:6:"handle";s:10:"2108/55871";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:73:"Ghrelin improves endothelial function in patients with metabolic syndrome";s:9:"metadata6";s:81:"Tesauro, M; Schinzari, F; Iantorno, M; Rizza, S; Melina, D; Lauro, D; Cardillo, C";s:9:"metadata7";s:33:"10.1161/CIRCULATIONAHA.105.553883";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:159;a:14:{s:9:"citazione";s:198:"Tesauro, M., Schinzari, F., Iantorno, M., Rizza, S., Melina, D., Lauro, D., et al. (2005). Ghrelin improves endothelial function in patients with metabolic syndrome. CIRCULATION, 112(19), 2986-2992.";s:4:"data";s:4:"2005";s:2:"id";s:20:"PUBBLICAZIONE_239613";s:6:"handle";s:11:"2108/120758";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:224:"Acetylcholine; Adult; Endothelium, Vascular; Female; Ghrelin; Glucose Intolerance; Humans; Male; Metabolic Syndrome X; Middle Aged; Nitroprusside; Obesity; Peptide Hormones; Reference Values; Vasodilation; Vasodilator Agents";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:391:"Metabolic syndrome importantly accelerates the atherosclerotic process, the earliest event of which is endothelial dysfunction. Ghrelin, a gastric peptide with cardiovascular actions, has been shown to inhibit proatherogenic changes in experimental models. This study therefore investigated whether ghrelin administration might beneficially affect endothelial function in metabolic syndrome.";s:9:"metadata5";s:73:"Ghrelin improves endothelial function in patients with metabolic syndrome";s:9:"metadata6";s:81:"Tesauro, M; Schinzari, F; Iantorno, M; Rizza, S; Melina, D; Lauro, D; Cardillo, C";s:9:"metadata7";s:33:"10.1161/CIRCULATIONAHA.105.553883";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:160;a:14:{s:9:"citazione";s:283:"Troisi, A., Di Lorenzo, G., Lega, I., Tesauro, M., Bertoli, A., Leo, R., et al. (2005). Plasma ghrelin in anorexia, bulimia, and binge-eating disorder: Relations with eating patterns and circulating concentrations of cortisol and thyroid hormones. NEUROENDOCRINOLOGY, 81(4), 259-266.";s:4:"data";s:4:"2005";s:2:"id";s:20:"PUBBLICAZIONE_239607";s:6:"handle";s:11:"2108/120752";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";s:180:"Adult; Anorexia; Body Mass Index; Bulimia; Female; Ghrelin; Humans; Hydrocortisone; Nutritional Status; Peptide Hormones; Thyroid Hormones; Thyrotropin; Thyroxine; Triiodothyronine";s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";s:1579:"The present study was designed to investigate the relations between plasma ghrelin concentrations, eating patterns, and circulating concentrations of cortisol and thyroid hormones in women with anorexia nervosa, bulimia nervosa, and binge-eating disorder. The patterns of disordered eating behavior were assessed using the Eating Attitudes Test (EAT-26) and the Bulimia Test-Revised (BULIT-R). In women with eating disorders, but not in healthy control women, plasma ghrelin concentrations were negatively correlated with body mass index (BMI) and plasma concentrations of thyreotropin (TSH), free T3 and free T4, and positively correlated with plasma concentrations of cortisol. The ghrelin concentrations of women with binge-eating and purging behavior were significantly lower than those of women with anorexia nervosa, restricting type, and there was a negative relation between the frequency and severity of binge-eating and purging behavior, as measured by the BULIT-R total score, and ghrelin concentrations. In a multivariate regression model controlling for the confounding effects of body mass index (BMI) and age, higher ghrelin concentrations were correlated with lower BULIT-R total scores. The results of this study did not confirm the hypothesis advanced in previous studies that ghrelin concentrations are higher in patients with binge-eating/purging forms of eating disorders. Based on these data, we suggest that, in women with eating disorders, ghrelin concentrations best reflect nutritional status rather than specific patterns of disordered eating behavior.";s:9:"metadata5";s:158:"Plasma ghrelin in anorexia, bulimia, and binge-eating disorder: Relations with eating patterns and circulating concentrations of cortisol and thyroid hormones";s:9:"metadata6";s:142:"Troisi, A; Di Lorenzo, G; Lega, I; Tesauro, M; Bertoli, A; Leo, R; Iantorno, M; Pecchioli, C; Rizza, S; Turriziani, M; Lauro, R; Siracusano, A";s:9:"metadata7";s:17:"10.1159/000087923";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:161;a:14:{s:9:"citazione";s:279:"D'Adamo, M., Perego, L., Cardellini, M., Marini, M.A., Frontoni, S., Andreozzi, F., et al. (2004). The -866A/A genotype in the promoter of the human uncoupling protein 2 gene is associated with insulin resistance and increased risk of type 2 diabetes. DIABETES, 53(7), 1905-1910.";s:4:"data";s:7:"2004-07";s:2:"id";s:19:"PUBBLICAZIONE_30794";s:6:"handle";s:10:"2108/33957";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:151:"The -866A/A genotype in the promoter of the human uncoupling protein 2 gene is associated with insulin resistance and increased risk of type 2 diabetes";s:9:"metadata6";s:197:"D'Adamo, M; Perego, L; Cardellini, M; Marini, MA; Frontoni, S; Andreozzi, F; Sciacqua, A; Lauro, D; Sbraccia, P; Federici, M; Paganelli, M; Pontiroli, AE; Lauro, R; Perticone, F; Folli, F; Sesti, G";s:9:"metadata7";s:26:"10.2337/diabetes.53.7.1905";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:162;a:14:{s:9:"citazione";s:300:"D'Alessandris, C., Andreozzi, F., Federici, M., Cardellini, M., Brunetti, A., Ranalli, M., et al. (2004). Increased O-glycosylation of insulin signaling proteins results in their impaired activation and enhanced susceptibility to apoptosis in pancreatic beta-cells. THE FASEB JOURNAL, 18(9), 959-961.";s:4:"data";s:7:"2004-06";s:2:"id";s:19:"PUBBLICAZIONE_90214";s:6:"handle";s:10:"2108/36448";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:158:"Increased O-glycosylation of insulin signaling proteins results in their impaired activation and enhanced susceptibility to apoptosis in pancreatic beta-cells";s:9:"metadata6";s:156:"D'Alessandris, C; Andreozzi, F; Federici, M; Cardellini, M; Brunetti, A; Ranalli, M; Del Guerra, S; Lauro, D; Del Prato, S; Marchetti, P; Lauro, R; Sesti, G";s:9:"metadata7";s:21:"10.1096/fj.03-0725fje";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:163;a:14:{s:9:"citazione";s:251:"Federici, M., Pandolfi, A., De Filippis, E., Pellegrini, G., Menghini, R., Lauro, D., et al. (2004). G972R IRS-1 variant impairs insulin regulation of endothelial nitric oxide synthase in cultured human endothelial cells. CIRCULATION, 109(3), 399-405.";s:4:"data";s:10:"2004-01-27";s:2:"id";s:19:"PUBBLICAZIONE_89413";s:6:"handle";s:10:"2108/34871";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:33:"Settore MED/09 - Medicina Interna";s:9:"metadata4";N;s:9:"metadata5";s:119:"G972R IRS-1 variant impairs insulin regulation of endothelial nitric oxide synthase in cultured human endothelial cells";s:9:"metadata6";s:136:"Federici, M; Pandolfi, A; De Filippis, E; Pellegrini, G; Menghini, R; Lauro, D; Cardellini, M; Romano, M; Sesti, G; Lauro, R; Consoli, A";s:9:"metadata7";s:34:"10.1161/01.CIR.0000109498.77895.6F";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:164;a:14:{s:9:"citazione";s:290:"Sesti, G., Marini, M.A., Cardellini, M., Sciacqua, A., Frontoni, S., Andreozzi, F., et al. (2004). The Arg972 variant in insulin receptor substrate-1 is associated with an increased risk of secondary failure to sulfonylurea in patients with type 2 diabetes. DIABETES CARE, 27(6), 1394-1398.";s:4:"data";s:4:"2004";s:2:"id";s:18:"PUBBLICAZIONE_4241";s:6:"handle";s:10:"2108/30969";s:9:"metadata1";s:19:"Articolo su rivista";s:9:"metadata2";N;s:9:"metadata3";s:31:"Settore MED/13 - Endocrinologia";s:9:"metadata4";N;s:9:"metadata5";s:157:"The Arg972 variant in insulin receptor substrate-1 is associated with an increased risk of secondary failure to sulfonylurea in patients with type 2 diabetes";s:9:"metadata6";s:143:"Sesti, G; Marini, MA; Cardellini, M; Sciacqua, A; Frontoni, S; Andreozzi, F; Irace, C; Lauro, D; Gnasso, A; Federici, M; Perticone, F; Lauro, R";s:9:"metadata7";s:25:"10.2337/diacare.27.6.1394";s:9:"metadata8";N;s:9:"metadata9";N;s:10:"metadata10";N;}i:165;a:14:{s:9:"citazione";s:297:"Hribal, M., Perego, L., Lovari, S., Andreozzi, F., Menghini, R., Perego, C., et al. (2003). Chronic hyperglycemia impairs insulin secretion by affecting insulin receptor expression, splicing, and signaling in RIN beta cell line and human islets of Langerhans. 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